Publicaciones en las que colabora con Iván Quesada Moll (27)

2023

  1. Acute and chronic effects of the organophosphate malathion on the pancreatic α and β cell viability, cell structure, and voltage-gated K+ currents.

    Environmental Toxicology and Pharmacology, Vol. 98

  2. Endocrine disruptors in plastics alter β-cell physiology and increase the risk of diabetes mellitus

    American journal of physiology. Endocrinology and metabolism, Vol. 324, Núm. 6, pp. E488-E505

  3. Exploring the Effects of Metabolism-Disrupting Chemicals on Pancreatic α-Cell Viability, Gene Expression and Function: A Screening Testing Approach

    International Journal of Molecular Sciences, Vol. 24, Núm. 2

2022

  1. The Effects of Aging on Male Mouse Pancreatic β-Cell Function Involve Multiple Events in the Regulation of Secretion: Influence of Insulin Sensitivity

    The journals of gerontology. Series A, Biological sciences and medical sciences, Vol. 77, Núm. 3, pp. 405-415

  2. The pancreatic β-cell in ageing: Implications in age-related diabetes

    Ageing Research Reviews, Vol. 80

2021

  1. Bisphenol-S and Bisphenol-F alter mouse pancreatic β-cell ion channel expression and activity and insulin release through an estrogen receptor ERβ mediated pathway

    Chemosphere, Vol. 265

2019

  1. Cortistatin regulates glucose-induced electrical activity and insulin secretion in mouse pancreatic beta-cells

    Molecular and Cellular Endocrinology, Vol. 479, pp. 123-132

  2. Oestrogen receptor β mediates the actions of bisphenol-A on ion channel expression in mouse pancreatic beta cells

    Diabetologia, Vol. 62, Núm. 9, pp. 1667-1680

2018

  1. Erratum: Author Correction: Molecular mechanisms involved in the non-monotonic effect of bisphenol-a on Ca2+ entry in mouse pancreatic β-cells (Scientific reports (2017) 7 1 (11770))

    Scientific reports

  2. Extranuclear-initiated estrogenic actions of endocrine disrupting chemicals: Is there toxicology beyond paracelsus?

    Journal of Steroid Biochemistry and Molecular Biology, Vol. 176, pp. 16-22

2017

  1. Molecular mechanisms involved in the non-monotonic effect of bisphenol-a on ca2+ entry in mouse pancreatic β-cells

    Scientific Reports, Vol. 7, Núm. 1

2016

  1. Effects of Bisphenol A on ion channels: Experimental evidence and molecular mechanisms

    Steroids, Vol. 111, pp. 12-20

  2. The bile acid TUDCA increases glucose-induced insulin secretion via the cAMP/PKA pathway in pancreatic beta cells

    Metabolism: Clinical and Experimental, Vol. 65, Núm. 3, pp. 54-63

2013

  1. Antidiabetic actions of an estrogen receptor β selective agonist

    Diabetes, Vol. 62, Núm. 6, pp. 2015-2025

  2. Insulin hypersecretion in islets from diet-induced hyperinsulinemic obese female mice is associated with several functional adaptations in individual β-cells

    Endocrinology, Vol. 154, Núm. 10, pp. 3515-3524

2012

  1. Bisphenol-A acts as a potent estrogen via non-classical estrogen triggered pathways

    Molecular and Cellular Endocrinology, Vol. 355, Núm. 2, pp. 201-207

  2. Rapid insulinotropic action of low doses of Bisphenol-A on mouse and human islets of Langerhans: Role of estrogen receptor β

    PLoS ONE, Vol. 7, Núm. 2

2011

  1. Regulation of KATP channel by 17β-estradiol in pancreatic β-cells

    Steroids, Vol. 76, Núm. 9, pp. 856-860

  2. Role of estrogen receptors alpha, beta and GPER1/GPR30 in pancreatic beta-cells

    Frontiers in Bioscience, Vol. 16, Núm. 1, pp. 251-260

2010

  1. Bisphenol a exposure during pregnancy disrupts glucose homeostasis in mothers and adult male offspring

    Environmental Health Perspectives, Vol. 118, Núm. 9, pp. 1243-1250