Publikationen, an denen er mitarbeitet Ángel Nadal Navajas (30)

2023

  1. Endocrine disruptors in plastics alter β-cell physiology and increase the risk of diabetes mellitus

    American journal of physiology. Endocrinology and metabolism, Vol. 324, Núm. 6, pp. E488-E505

2022

  1. G protein-coupled estrogen receptor activation by bisphenol-A disrupts the protection from apoptosis conferred by the estrogen receptors ERα and ERβ in pancreatic beta cells

    Environment International, Vol. 164

  2. The Effects of Aging on Male Mouse Pancreatic β-Cell Function Involve Multiple Events in the Regulation of Secretion: Influence of Insulin Sensitivity

    The journals of gerontology. Series A, Biological sciences and medical sciences, Vol. 77, Núm. 3, pp. 405-415

  3. The pancreatic β-cell in ageing: Implications in age-related diabetes

    Ageing Research Reviews, Vol. 80

2021

  1. Bisphenol-S and Bisphenol-F alter mouse pancreatic β-cell ion channel expression and activity and insulin release through an estrogen receptor ERβ mediated pathway

    Chemosphere, Vol. 265

2019

  1. Bisphenol A Regulates Sodium Ramp Currents in Mouse Dorsal Root Ganglion Neurons and Increases Nociception

    Scientific Reports, Vol. 9, Núm. 1

  2. Cortistatin regulates glucose-induced electrical activity and insulin secretion in mouse pancreatic beta-cells

    Molecular and Cellular Endocrinology, Vol. 479, pp. 123-132

  3. Oestrogen receptor β mediates the actions of bisphenol-A on ion channel expression in mouse pancreatic beta cells

    Diabetologia, Vol. 62, Núm. 9, pp. 1667-1680

2018

  1. A calcium-dependent chloride current increases repetitive firing in mouse sympathetic neurons

    Frontiers in Physiology, Vol. 9, Núm. MAY

  2. Erratum: Author Correction: Molecular mechanisms involved in the non-monotonic effect of bisphenol-a on Ca2+ entry in mouse pancreatic β-cells (Scientific reports (2017) 7 1 (11770))

    Scientific reports

  3. Extranuclear-initiated estrogenic actions of endocrine disrupting chemicals: Is there toxicology beyond paracelsus?

    Journal of Steroid Biochemistry and Molecular Biology, Vol. 176, pp. 16-22

2017

  1. Molecular mechanisms involved in the non-monotonic effect of bisphenol-a on ca2+ entry in mouse pancreatic β-cells

    Scientific Reports, Vol. 7, Núm. 1

2016

  1. Effects of Bisphenol A on ion channels: Experimental evidence and molecular mechanisms

    Steroids, Vol. 111, pp. 12-20

  2. The bile acid TUDCA increases glucose-induced insulin secretion via the cAMP/PKA pathway in pancreatic beta cells

    Metabolism: Clinical and Experimental, Vol. 65, Núm. 3, pp. 54-63

2013

  1. Antidiabetic actions of an estrogen receptor β selective agonist

    Diabetes, Vol. 62, Núm. 6, pp. 2015-2025

  2. Insulin hypersecretion in islets from diet-induced hyperinsulinemic obese female mice is associated with several functional adaptations in individual β-cells

    Endocrinology, Vol. 154, Núm. 10, pp. 3515-3524

2012

  1. Bisphenol-A acts as a potent estrogen via non-classical estrogen triggered pathways

    Molecular and Cellular Endocrinology, Vol. 355, Núm. 2, pp. 201-207

  2. Insulinotropic effect of the non-steroidal compound STX in pancreatic β-cells

    PLoS ONE, Vol. 7, Núm. 4

  3. Rapid insulinotropic action of low doses of Bisphenol-A on mouse and human islets of Langerhans: Role of estrogen receptor β

    PLoS ONE, Vol. 7, Núm. 2

2011

  1. Involvement of ATP-sensitive potassium (K ATP) channels in the loss of beta-cell function induced by human islet amyloid polypeptide

    Journal of Biological Chemistry, Vol. 286, Núm. 47, pp. 40857-40866